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Perspective - (2023) Volume 8, Issue 1

Glyconanoparticles that are Specifically Linked to Stomach and Digestive Tract Disease
Zikai Lin*
Department of Radio Oncology, University of Aveiro, Portugal
*Correspondence: Zikai Lin, Department of Radio Oncology, University of Aveiro, Portugal, Email:

Received: 31-Jan-2023, Manuscript No. IPJCEP-23-16271; Editor assigned: 02-Feb-2023, Pre QC No. IPJCEP-23-16271 (PQ); Reviewed: 16-Feb-2023, QC No. IPJCEP-23-16271; Revised: 21-Feb-2023, Manuscript No. IPJCEP-23-16271 (R); Published: 28-Feb-2023, DOI: 10.36648/IPJCEP.23.08.009


When a cell lacks the ability to repair DNA damage, it tends to keep it at a higher level. During the process of the cell’s DNA replication, these issues may result in errors, including changes that cause disease. Numerous inherited DNA repair disorders have been linked to an increased risk of cancer. Moreover, various tumors need specific DNA fix chemicals. For instance, numerous malignancies have been linked to low levels of the DNA fixative O-6-methylguanine-DNA methyltransferase.


According to Lingg and colleagues, cancer cell lineages may show an increase in the expression of a repair capability, which may be of clinical significance as they develop. A cell lineage may be more likely to develop cancer if it does not properly repair DNA. A protein that is typically only expressed in meiotic cells, where it maintains an unaffected organism line, is encoded by the DNA fix quality DMC1. Be that as it may, various illness cell lines, like lymphoma, cervical, and chest dangerous development cell lines, express DMC1. Meiotic DNA fix characteristics like DMC1 can possibly both speed up the advancement of disease cells and lessen the practicality of anticancer therapies like radiation treatment by controlling endogenous DNA harm during the development interaction. Incidental effects may occur nearby due to the disease’s mass or ulceration. The block of the bronchi, which can bring about pneumonia or hacking, is one of the many impacts of cellular breakdown in the lungs. The throat may become congested as a result of an esophageal malignant growth, making gulping difficult or painful. Additionally, bowel narrowing or obstruction caused by colorectal cancer can alter bowel habits. There can be noticeable knots caused by masses in the gonads or stomach. Ulcerative depletion can lead to secondary effects like bleeding in the stool, weakness or rectal death, blood in the urine, and strange vaginal passing. Patients with state of the art sickness could experience confined torture, yet the hidden development consistently causes no irritation. Liquid biopsy, otherwise called CTC acknowledgment, enjoys various upper hands over ordinary tissue biopsies. They are easy to use, can be used multiple times, and provide more important information regarding the progression of the disease, the viability of treatment, and metastatic disease. For instance, the progression of malignant growth has been linked to the likelihood of CTC identification in blood tests. The procedure is simple and safe, and multiple blood samples can be taken at the same time. It seems paradoxical that intrusive methods that may limit patient consistency are required for the investigation of strong cancers. It may be possible to work on a patient’s visualization and personal satisfaction by simplifying the process of changing their treatment when it is possible to follow the progression of the infection over time. When repeated CTC counts are low and do not rise, it is essential to avoid the need for surgery in order to predict how the disease will progress in the future; Avoiding the bet that goes with sickness operations’ natural malignant growth genicity is one of the obvious advantages of avoiding an operation.


Cell breakdown in the lungs is by and large consistently achieved by smoking. The remaining cases were caused by accidental genetic mutations or exposure to hazardous substances like radon gas and asbestos. As a result, efforts to stop cell breakdown in the lungs encourage people to avoid harmful engineered compounds and quit smoking. Quitting smoking reduces the risk of spreading the disease and improves treatment outcomes for lung cancer patients with cellular breakdown.



Conflict of Interest

The authors declare that they have no conflict of interest.

Citation: Lin Z (2023) Glyconanoparticles that are Specifically Linked to Stomach and Digestive Tract Disease. J Cancer Epidemiol Prev. 8:009.

Copyright: © 2023 Lin Z. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.